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Adolescents in Taiwan: the spread was bigger than the result

Forty-one adolescents lost 4.4 kg on average over 26 weeks. The standard deviation was 5.7, so the scatter included teenagers who gained.

Wesley Jenkins7 min read
Weight change, mean and one standard deviationno changeweek 13-2.7 ± 3.8 kgweek 26-4.4 ± 5.7 kgBoth bands cross zero. Some of these teenagers gained.

Two numbers are printed beside every average in this paper. Most readers will take the first. The second is the more useful one.

What the averages hide

At week 26 the group had lost 4.4 kg. The standard deviation was 5.7 kg. [1] At week 13 it was 2.7 kg lost, with a deviation of 3.8.

A spread wider than the mean means the results ran in both directions. Some of these adolescents lost a great deal. Others gained while on the drug. The average is real, and nobody in the study is average.

That matters more for a family deciding whether to pay than the headline does. A figure like this is not a forecast. It is the middle of a wide scatter, and early response varies enormously even in the trials.

Who was in it

Forty-one adolescents, mean age 13.9, mean weight 88.5 kg, mean BMI 33.4. Just over half were boys. Nineteen of them — 46.3% — had a history of hepatic impairment.

That last figure is worth noticing. It is a much sicker sample than a general adolescent obesity population, and it tells you something about who gets referred for this in Taiwan.

No control group

Everybody got the drug. There is nothing to compare against.

Twenty-six weeks in a clinic supplies more than an injection. It supplies appointments, weigh-ins, advice and somebody paying attention. Adolescents also grow, which is why the study reports a BMI standard deviation score alongside raw weight. None of that separates on its own, and it is the same difficulty as isolating which part of an effect came from the drug.

The safety half

Adverse events reached 19.5%, eight of forty-one, all mild. Drug reactions were 9.8%, four people and six events. Gastrointestinal complaints led, then injection-site reactions.

Nobody stopped because they could not tolerate it. In a group this small that is encouraging and not much more — forty-one people cannot rule out anything uncommon.

If this is your decision

It belongs with a pediatric specialist, not a website. Dosing in a growing adolescent with liver involvement is not a comparison-shopping question.

What this page can tell you is how to read the number when you meet it. Four kilograms over six months, at half the licensed dose, in a scatter wide enough to include people who gained — and the price of those six months is the same whichever end of the scatter you land on, which is the part every cost-effectiveness model has to assume away.

Frequently asked

How much weight did these adolescents lose?
An average of 2.7 kg by week 13 and 4.4 kg by week 26. The standard deviations were 3.8 and 5.7 kg, both larger than the average.
Why does the standard deviation matter?
Because it is wider than the mean, which means the results ran in both directions. Some participants lost a great deal and others gained while taking the drug.
Was this the full dose?
No. The licensed obesity dose of liraglutide is 3.0 mg a day. The mean daily dose here was 1.4 mg, and 14.4% of participants ever reached 3.0.
Were there side effects?
Adverse events occurred in 19.5%, all mild, and nobody discontinued because of them. Forty-one people is too few to rule out anything uncommon.

Sources

  1. [1] Chiu CF, et al. (2026). A multicenter real-world study on the safety and effectiveness of the glucagon-like peptide-1 receptor agonist (liraglutide) in adolescents with obesity in Taiwan Obesity Research & Clinical Practice. PMID 42744726

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